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Investigating the role of saposin B in HMA-mediated toxicity toward breast cancer cells
The data associated with this publication are not available for this reason: N/A
Abstract
Therapy-resistant cancer cells can be targeted using novel cationic amphiphilic drugs such as HMA that induce lysosomal cell death. Parallel alteration of lysosomal protein Saposin B could sensitize cells to HMA, reducing off-target effects.
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