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That Wasn’t a Complement—Too Much C3 in Demyelinating Disease
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https://doi.org/10.1016/j.immuni.2019.12.014Abstract
Multiple sclerosis is a chronic inflammatory disease characterized by demyelination in the central nervous system. In this issue of Immunity, Werneberg et al. report a striking loss of synapses driven by excessive microglial pruning early in demyelinating disease, which can be rescued by inhibiting the complement component C3.
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