Skip to main content
Variety in the Tumor Microenvironment: Integrin Splicing Regulates Stemness
Published Web Location
http://10.0.3.248/j.stem.2014.04.011No data is associated with this publication.
Abstract
Recently in Cell Reports, Goel et al. (2014) identified mechanisms underlying cellular heterogeneity in triple negative breast cancer. They find that expression of α6 integrin and its splice variants differs between epithelial and mesenchymal tumor cell subpopulations, the latter of which relies on VEGF signaling to promote cancer stem cell function.
Many UC-authored scholarly publications are freely available on this site because of the UC's open access policies. Let us know how this access is important for you.