To reveal the effects of Co-doping on the electrochemical performance of micro-sized LiNi0.5Mn1.5O4 (LNMO), undoped LNMO and Co-doped LiCo0.1Ni0.45Mn1.45O4 (LCoNMO) are synthesized via a PVP-combustion method and calcined at 1000 °C for 6 h. SEM and XRD analyses suggest that Co-doping decreases the particle size and the LizNi1-zO2 impurity at the calcination temperature of 1000 °C. LCoNMO has much better rate capability while its specific capacity at C/5 is 10% lower than that of LNMO. At 15 C rate, their specific capacities are closed, and the LCoNMO delivers 86.2% capacity relative to C/5, and this value for LNMO is only 77.0%. The DLi + values determined by potential intermittent titration technique (PITT) test of LCoNMO are 1-2 times higher than that of LNMO in most SOC region. The LCoNMO shows very excellent cycling performance, which is the best value compared with literatures. After 1000 cycles, the LCoNMO still delivers 94.1% capacity. Moreover, its coulombic efficiency and energy efficiency keep at 99.84% and over 97.3% during 1 C cycling, respectively.

Rhythmic intraorgan communication coordinates environmental signals and the cell-intrinsic clock to maintain organ homeostasis. Hepatocyte-specific KO of core components of the molecular clock Rev-erbα and -β (Reverb-hDKO) alters cholesterol and lipid metabolism in hepatocytes as well as rhythmic gene expression in nonparenchymal cells (NPCs) of the liver. Here, we report that in fatty liver caused by diet-induced obesity (DIO), hepatocyte SREBP cleavage-activating protein (SCAP) was required for Reverb-hDKO-induced diurnal rhythmic remodeling and epigenomic reprogramming in liver macrophages (LMs). Integrative analyses of isolated hepatocytes and LMs revealed that SCAP-dependent lipidomic changes in REV-ERB-depleted hepatocytes led to the enhancement of LM metabolic rhythms. Hepatocytic loss of REV-ERBα and β (REV-ERBs) also attenuated LM rhythms via SCAP-independent polypeptide secretion. These results shed light on the signaling mechanisms by which hepatocytes regulate diurnal rhythms in NPCs in fatty liver disease caused by DIO.

A small in-plane external uniaxial pressure has been widely used as an effective method to acquire single domain iron pnictide BaFe₂As₂, which exhibits twin-domains without uniaxial strain below the tetragonal-to-orthorhombic structural (nematic) transition temperature T_s. Although it is generally assumed that such a pressure will not affect the intrinsic electronic/magnetic properties of the system, it is known to enhance the antiferromagnetic (AF) ordering temperature T_N ( < T_s) and create in-plane resistivity anisotropy above T_s. Here we use neutron polarization analysis to show that such a strain on BaFe₂As₂ also induces a static or quasi-static out-of-plane (c-axis) AF order and its associated critical spin fluctuations near T_N/T_s. Therefore, uniaxial pressure necessary to detwin single crystals of BaFe₂As₂ actually rotates the easy axis of the collinear AF order near T_N/T_s, and such effects due to spin-orbit coupling must be taken into account to unveil the intrinsic electronic/magnetic properties of the system.