- Malloy, Mary;
- Kane, John;
- Hossain, GS;
- Lynn, EG;
- Maclean, KN;
- Zhou, J;
- Dickhout, JG;
- Lhot́ak, S;
- Trigatti, B;
- Capone, J;
- Rho, J;
- Tang, D
Background: Apoptosis caused by endoplasmic reticulum (ER) stress contributes to atherothrombosis, the underlying cause of cardiovascular disease (CVD). T-cell death-associated gene 51 (TDAG51), a member of the pleckstrin homology-like domain gene family,
