A racial priming article claimed that, relative to a controlcondition, an exotic variety of variability, called 1/ƒ noise, isaltered when stereotypes impact participants’ judgments in animplicit prejudice task (Correll, 2008). However, Madurskiand LeBel (2014) recently described two powerful, faithfullycloned, and apparently decisive studies that each failed toreturn a successful literal replication of Correll’s report.Madurski and LeBel outlined and subsequently eliminatedseveral potential extraneous reasons for their replicationfailures, such as different participant demographics,participant non-compliance, poor psychometrics, andhardware discrepancies. By contrast, this article reports asuccessful conceptual replication of the pattern reported byCorrell (cf. Schmidt, 2009). Notably, this conceptualreplication required adjustments to Correll’s original methodand statistical analyses. All the changes were dictated by asystems theory of 1/ƒ noise that was largely in place prior toCorrell’s report (Kello, Beltz, Holden, & Van Orden, 2007;Van Orden, Holden, & Turvey, 2003; 2005). Implications forthe replication debate are discussed, with emphasis oncontextualizing implicit cues.

Cardiovascular autonomic dysfunction, such as orthostatic hypotension consequent to baroreflex failure and cardiac sympathetic denervation, is frequently observed in the synucleinopathy Parkinson's disease (PD). In the present study, the baroreceptor reflex was assessed in mice overexpressing human wildtype alpha-synuclein (Thy1-aSyn), a genetic mouse model of synucleinopathy. The beat-to-beat change in heart rate (HR), computed from R-R interval, in relation to blood pressure was measured in anesthetized and conscious mice equipped with arterial blood pressure telemetry transducers during transient bouts of hypertension and hypotension. Compared to wildtype, tachycardia following nitroprusside-induced hypotension was significantly reduced in Thy1-aSyn mice. Thy1-aSyn mice also showed an abnormal cardiovascular response (i.e., diminished tachycardia) to muscarinic blockade with atropine. We conclude that Thy1-aSyn mice have impaired basal and dynamic range of sympathetic and parasympathetic-mediated changes in HR and will be a useful model for long-term study of cardiovascular autonomic dysfunction associated with PD.